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Erythrocytosis has been reported as a possible impact of testosterone, which can be useful in sufferers with anemia. The biggest danger of erythrocytosis happens with injectable types of testosterone compared with transdermal preparations, particularly in supraphysiologic doses. An enhance of up to 44% of the baseline hematocrit has been reported .
Moreover, a element of endothelial dysfunction has been proposed, which can contribute to abnormal vessel reactivity. However, testosterone in physiologic doses may even be helpful in sufferers with angina , have higher ischemic thresholds and improved high quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and will increase coronary blood circulate in males with established coronary artery disease . Therefore, an interaction of endogenous elements of the patient’s risk profile in addition to the testosterone dosing may be issues of whether or not antagonistic events occur.
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- Men with coronary artery disease have decrease levels of androgens than men with normal coronary angiograms.
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We report a case of an acute ST-segment elevation myocardial infarction in a patient with traditional cardiac danger factors utilizing supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this affected person additionally had polycythemia, doubtless secondary to high-dose testosterone. The patient underwent successful percutaneous intervention of the right cqmsoda coronary artery. Physicians and sufferers ought to be aware of the clinical penalties of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).
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Rhoden EL, Morgentaler A. Risks of testosterone-replacement therapy and recommendations for monitoring. Testosterone alternative in hypogonadal men with angina improves ischaemic threshold and quality of life. Ajayi AAL, Mathur R, Halushka PV. Testosterone improve human platelet thromboxane A2 receptor density and aggregation responses.